Tham khảo Acid gamma-aminobutyric

↑ Haynes, William M., ed. (2016). CRC Handbook of Chemistry and Physics (97th ed.). CRC Press. p. 5–88. ISBN 978-1498754286.
↑ Watanabe M, Maemura K, Kanbara K, Tamayama T, Hayasaki H (2002). “GABA and GABA receptors in the central nervous system and other organs”. Trong Jeon KW (biên tập). Int. Rev. Cytol. International Review of Cytology. 213. tr. 1–47. doi:10.1016/S0074-7696(02)13011-7. ISBN 978-0-12-364617-0. PMID 11837891.
↑ Generalized Non-Convulsive Epilepsy: Focus on GABA-B Receptors, C. Marescaux, M. Vergnes, R. Bernasconi
↑ Ffrench-Constant RH, Rocheleau TA, Steichen JC, Chalmers AE (tháng 6 năm 1993). “A point mutation in a Drosophila GABA receptor confers insecticide resistance”. Nature. 363 (6428): 449–51. Bibcode:1993Natur.363..449F. doi:10.1038/363449a0. PMID 8389005.
↑ Szabadics J, Varga C, Molnár G, Oláh S, Barzó P, Tamás G (tháng 1 năm 2006). “Excitatory effect of GABAergic axo-axonic cells in cortical microcircuits”. Science. 311 (5758): 233–235. Bibcode:2006Sci...311..233S. doi:10.1126/science.1121325. PMID 16410524.
1 2 Li K, Xu E (tháng 6 năm 2008). “The role and the mechanism of γ-aminobutyric acid during central nervous system development”. Neurosci Bull. 24 (3): 195–200. doi:10.1007/s12264-008-0109-3. PMC 5552538. PMID 18500393.
↑ Ben-Ari Y, Gaiarsa JL, Tyzio R, Khazipov R (tháng 10 năm 2007). “GABA: a pioneer transmitter that excites immature neurons and generates primitive oscillations”. Physiol. Rev. 87 (4): 1215–1284. doi:10.1152/physrev.00017.2006. PMID 17928584.
↑ The Glutamate/GABA-Glutamine Cycle: Amino Acid Neurotransmitter Homeostasis, Arne Schousboe, Ursula Sonnewald
↑ Purves D, Fitzpatrick D, Hall WC, Augustine GJ, Lamantia AS biên tập (2007). Neuroscience (ấn bản 4). Sunderland, Mass: Sinauer. tr. 135, box 6D. ISBN 978-0-87893-697-7.
↑ Jelitai M, Madarasz E (2005). The role of GABA in the early neuronal development. Int. Rev. Neurobiol. International Review of Neurobiology. 71. tr. 27–62. doi:10.1016/S0074-7742(05)71002-3. ISBN 9780123668721. PMID 16512345.
↑ Marín O, Rubenstein JL (tháng 11 năm 2001). “A long, remarkable journey: tangential migration in the telencephalon”. Nat. Rev. Neurosci. 2 (11): 780–90. doi:10.1038/35097509. PMID 11715055.
↑ LoTurco JJ, Owens DF, Heath MJ, Davis MB, Kriegstein AR (tháng 12 năm 1995). “GABA and glutamate depolarize cortical progenitor cells and inhibit DNA synthesis”. Neuron. 15 (6): 1287–1298. doi:10.1016/0896-6273(95)90008-X. PMID 8845153.
↑ Haydar TF, Wang F, Schwartz ML, Rakic P (tháng 8 năm 2000). “Differential modulation of proliferation in the neocortical ventricular and subventricular zones”. J. Neurosci. 20 (15): 5764–74. doi:10.1523/JNEUROSCI.20-15-05764.2000. PMC 3823557. PMID 10908617.
↑ Behar TN, Schaffner AE, Scott CA, O'Connell C, Barker JL (tháng 8 năm 1998). “Differential response of cortical plate and ventricular zone cells to GABA as a migration stimulus”. J. Neurosci. 18 (16): 6378–87. doi:10.1523/JNEUROSCI.18-16-06378.1998. PMID 9698329.
↑ Ganguly K, Schinder AF, Wong ST, Poo M (tháng 5 năm 2001). “GABA itself promotes the developmental switch of neuronal GABAergic responses from excitation to inhibition”. Cell. 105 (4): 521–32. doi:10.1016/S0092-8674(01)00341-5. PMID 11371348.
↑ Barbin G, Pollard H, Gaïarsa JL, Ben-Ari Y (tháng 4 năm 1993). “Involvement of GABAA receptors in the outgrowth of cultured hippocampal neurons”. Neurosci. Lett. 152 (1–2): 150–154. doi:10.1016/0304-3940(93)90505-F. PMID 8390627.
↑ Maric D, Liu QY, Maric I, Chaudry S, Chang YH, Smith SV, Sieghart W, Fritschy JM, Barker JL (tháng 4 năm 2001). “GABA expression dominates neuronal lineage progression in the embryonic rat neocortex and facilitates neurite outgrowth via GABA(A) autoreceptor/Cl− channels”. J. Neurosci. 21 (7): 2343–60. doi:10.1523/JNEUROSCI.21-07-02343.2001. PMID 11264309.
↑ Ben-Ari Y (tháng 9 năm 2002). “Excitatory actions of gaba during development: the nature of the nurture”. Nat. Rev. Neurosci. 3 (9): 728–739. doi:10.1038/nrn920. PMID 12209121.
↑ Obrietan K, Gao XB, Van Den Pol AN (tháng 8 năm 2002). “Excitatory actions of GABA increase BDNF expression via a MAPK-CREB-dependent mechanism—a positive feedback circuit in developing neurons”. J. Neurophysiol. 88 (2): 1005–15. doi:10.1152/jn.2002.88.2.1005. PMID 12163549.
↑ Wang DD, Kriegstein AR, Ben-Ari Y (2008). “GABA regulates stem cell proliferation before nervous system formation”. Epilepsy Curr. 8 (5): 137–9. doi:10.1111/j.1535-7511.2008.00270.x. PMC 2566617. PMID 18852839.
↑ Popp A, Urbach A, Witte OW, Frahm C (2009). Reh TA (biên tập). “Adult and embryonic GAD transcripts are spatiotemporally regulated during postnatal development in the rat brain”. PLoS ONE. 4 (2): e4371. Bibcode:2009PLoSO...4.4371P. doi:10.1371/journal.pone.0004371. PMC 2629816. PMID 19190758.
↑ Rorsman P, Berggren PO, Bokvist K, Ericson H, Möhler H, Ostenson CG, Smith PA (1989). “Glucose-inhibition of glucagon secretion involves activation of GABAA-receptor chloride channels”. Nature. 341 (6239): 233–6. Bibcode:1989Natur.341..233R. doi:10.1038/341233a0. PMID 2550826.
1 2 Soltani N, Qiu H, Aleksic M, Glinka Y, Zhao F, Liu R, Li Y, Zhang N, Chakrabarti R, Ng T, Jin T, Zhang H, Lu WY, Feng ZP, Prud'homme GJ, Wang Q (2011). “GABA exerts protective and regenerative effects on islet beta cells and reverses diabetes”. Proc. Natl. Acad. Sci. U.S.A. 108 (28): 11692–7. Bibcode:2011PNAS..10811692S. doi:10.1073/pnas.1102715108. PMC 3136292. PMID 21709230.
↑ Tian J, Dang H, Chen Z, Guan A, Jin Y, Atkinson MA, Kaufman DL (2013). “γ-Aminobutyric acid regulates both the survival and replication of human β-cells”. Diabetes. 62 (11): 3760–5. doi:10.2337/db13-0931. PMC 3806626. PMID 23995958.
↑ Purwana I, Zheng J, Li X, Deurloo M, Son DO, Zhang Z, Liang C, Shen E, Tadkase A, Feng ZP, Li Y, Hasilo C, Paraskevas S, Bortell R, Greiner DL, Atkinson M, Prud'homme GJ, Wang Q (2014). “GABA promotes human β-cell proliferation and modulates glucose homeostasis”. Diabetes. 63 (12): 4197–205. doi:10.2337/db14-0153. PMID 25008178.
↑ Ben-Othman N, Vieira A, Courtney M, Record F, Gjernes E, Avolio F, Hadzic B, Druelle N, Napolitano T, Navarro-Sanz S, Silvano S, Al-Hasani K, Pfeifer A, Lacas-Gervais S, Leuckx G, Marroquí L, Thévenet J, Madsen OD, Eizirik DL, Heimberg H, Kerr-Conte J, Pattou F, Mansouri A, Collombat P (2017). “Long-Term GABA Administration Induces Alpha Cell-Mediated Beta-like Cell Neogenesis”. Cell. 168 (1–2): 73–85.e11. doi:10.1016/j.cell.2016.11.002. PMID 27916274.
↑ Erdö SL, Wolff JR (tháng 2 năm 1990). “γ-Aminobutyric acid outside the mammalian brain”. J. Neurochem. 54 (2): 363–72. doi:10.1111/j.1471-4159.1990.tb01882.x. PMID 2405103.
↑ Tian J, Chau C, Hales TG, Kaufman DL (1999). “GABAA receptors mediate inhibition of T cell responses”. J. Neuroimmunol. 96 (1): 21–8. doi:10.1016/s0165-5728(98)00264-1. PMID 10227421.
↑ Mendu SK, Bhandage A, Jin Z, Birnir B (2012). “Different subtypes of GABA-A receptors are expressed in human, mouse and rat T lymphocytes”. PLoS ONE. 7 (8): e42959. Bibcode:2012PLoSO...742959M. doi:10.1371/journal.pone.0042959. PMC 3424250. PMID 22927941.
↑ Tian J, Lu Y, Zhang H, Chau CH, Dang HN, Kaufman DL (2004). “Gamma-aminobutyric acid inhibits T cell autoimmunity and the development of inflammatory responses in a mouse type 1 diabetes model”. J. Immunol. 173 (8): 5298–304. doi:10.4049/jimmunol.173.8.5298. PMID 15470076.
↑ Tian J, Yong J, Dang H, Kaufman DL (2011). “Oral GABA treatment downregulates inflammatory responses in a mouse model of rheumatoid arthritis”. Autoimmunity. 44 (6): 465–70. doi:10.3109/08916934.2011.571223. PMC 5787624. PMID 21604972.
↑ Bhandage AK, Jin Z, Korol SV, Shen Q, Pei Y, Deng Q, Espes D, Carlsson PO, Kamali-Moghaddam M, Birnir B (tháng 4 năm 2018). “+ T Cells and Is Immunosuppressive in Type 1 Diabetes”. EBioMedicine. 30: 283–294. doi:10.1016/j.ebiom.2018.03.019. PMC 5952354. PMID 29627388.
↑ Xiang YY, Wang S, Liu M, Hirota JA, Li J, Ju W, Fan Y, Kelly MM, Ye B, Orser B, O'Byrne PM, Inman MD, Yang X, Lu WY (tháng 7 năm 2007). “A GABAergic system in airway epithelium is essential for mucus overproduction in asthma”. Nat. Med. 13 (7): 862–7. doi:10.1038/nm1604. PMID 17589520.
↑ Payne AH, Hardy MH (2007). The Leydig cell in health and disease. Humana Press. ISBN 978-1-58829-754-9.
↑ Kwakowsky A, Schwirtlich M, Zhang Q, Eisenstat DD, Erdélyi F, Baranyi M, Katarova ZD, Szabó G (tháng 12 năm 2007). “GAD isoforms exhibit distinct spatiotemporal expression patterns in the developing mouse lens: correlation with Dlx2 and Dlx5”. Dev. Dyn. 236 (12): 3532–44. doi:10.1002/dvdy.21361. PMID 17969168.
↑ Ramesh SA, Tyerman SD, Xu B, Bose J, Kaur S, Conn V, Domingos P, Ullah S, Wege S, Shabala S, Feijó JA, Ryan PR, Gilliham M, Gillham M (2015). “GABA signalling modulates plant growth by directly regulating the activity of plant-specific anion transporters”. Nat Commun. 6: 7879. Bibcode:2015NatCo...6.7879R. doi:10.1038/ncomms8879. PMC 4532832. PMID 26219411.
↑ Ramesh SA, Tyerman SD, Gilliham M, Xu B (2016). “γ-Aminobutyric acid (GABA) signalling in plants”. Cell. Mol. Life Sci. 74 (9): 1577–1603. doi:10.1007/s00018-016-2415-7. PMID 27838745.
↑ Majumdar D, Guha S (1988). “Conformation, electrostatic potential and pharmacophoric pattern of GABA (γ-aminobutyric acid) and several GABA inhibitors”. Journal of Molecular Structure: THEOCHEM. 180: 125–140. doi:10.1016/0166-1280(88)80084-8.
1 2 Roth RJ, Cooper JR, Bloom FE (2003). The Biochemical Basis of Neuropharmacology. Oxford [Oxfordshire]: Oxford University Press. tr. 106. ISBN 978-0-19-514008-8.
↑ W. G. Van der Kloot; J. Robbins (1959). “The effects of GABA and picrotoxin on the junctional potential and the contraction of crayfish muscle”. Experientia. 15: 36.
↑ Petroff OA (tháng 12 năm 2002). “GABA and glutamate in the human brain”. Neuroscientist. 8 (6): 562–573. doi:10.1177/1073858402238515. PMID 12467378.
↑ Schousboe A, Waagepetersen HS (2007). GABA: homeostatic and pharmacological aspects. Prog. Brain Res. Progress in Brain Research. 160. tr. 9–19. doi:10.1016/S0079-6123(06)60002-2. ISBN 978-0-444-52184-2. PMID 17499106.
↑ Krantis, Anthony (ngày 1 tháng 12 năm 2000). “GABA in the Mammalian Enteric Nervous System”. Physiology. 15 (6): 284–290. doi:10.1152/physiologyonline.2000.15.6.284. ISSN 1548-9213.
↑ Sequerra, E. B.; Gardino, P.; Hedin-Pereira, C.; de Mello, F. G. (ngày 11 tháng 5 năm 2007). “Putrescine as an important source of GABA in the postnatal rat subventricular zone”. Neuroscience. 146 (2): 489–493. doi:10.1016/j.neuroscience.2007.01.062. ISSN 0306-4522. PMID 17395389.
1 2 Kuriyama K, Sze PY (tháng 1 năm 1971). “Blood–brain barrier to H3-γ-aminobutyric acid in normal and amino oxyacetic acid-treated animals”. Neuropharmacology. 10 (1): 103–108. doi:10.1016/0028-3908(71)90013-X. PMID 5569303.
↑ Boonstra E, de Kleijn R, Colzato LS, Alkemade A, Forstmann BU, Nieuwenhuis S (2015). “Neurotransmitters as food supplements: the effects of GABA on brain and behavior”. Front Psychol. 6: 1520. doi:10.3389/fpsyg.2015.01520. PMC 4594160. PMID 26500584.
↑ Bown AW, Shelp BJ (tháng 9 năm 1997). “The Metabolism and Functions of γ-Aminobutyric Acid”. Plant Physiol. 115 (1): 1–5. doi:10.1104/pp.115.1.1. PMC 158453. PMID 12223787.
↑ Foster AC, Kemp JA (tháng 2 năm 2006). “Glutamate- and GABA-based CNS therapeutics”. Curr Opin Pharmacol. 6 (1): 7–17. doi:10.1016/j.coph.2005.11.005. PMID 16377242.
↑ Chapouthier G, Venault P (tháng 10 năm 2001). “A pharmacological link between epilepsy and anxiety?”. Trends Pharmacol. Sci. 22 (10): 491–3. doi:10.1016/S0165-6147(00)01807-1. PMID 11583788.
↑ Campagna JA, Miller KW, Forman SA (tháng 5 năm 2003). “Mechanisms of actions of inhaled anesthetics”. N. Engl. J. Med. 348 (21): 2110–24. doi:10.1056/NEJMra021261. PMID 12761368.
↑ Powers ME, Yarrow JF, McCoy SC, Borst SE (tháng 1 năm 2008). “Growth hormone isoform responses to GABA ingestion at rest and after exercise”. Medicine and Science in Sports and Exercise. 40 (1): 104–10. doi:10.1249/mss.0b013e318158b518. PMID 18091016.
↑ Müller EE, Locatelli V, Cocchi D (tháng 4 năm 1999). “Neuroendocrine control of growth hormone secretion”. Physiol. Rev. 79 (2): 511–607. doi:10.1152/physrev.1999.79.2.511. PMID 10221989.
↑ Balemans MG, Mans D, Smith I, Van Benthem J (1983). “The influence of GABA on the synthesis of N-acetylserotonin, melatonin, O-acetyl-5-hydroxytryptophol and O-acetyl-5-methoxytryptophol in the pineal gland of the male Wistar rat”. Reproduction, Nutrition, Development. 23 (1): 151–60. doi:10.1051/rnd:19830114. PMID 6844712.
↑ Sato S, Yinc C, Teramoto A, Sakuma Y, Kato M (2008). “Sexually dimorphic modulation of GABA(A) receptor currents by melatonin in rats gonadotropin–releasing hormone neurons”. J Physiol Sci. 58 (5): 317–322. doi:10.2170/physiolsci.rp006208. PMID 18834560.
↑ The Brain, the Nervous System, and Their Diseases [3 volumes], Jennifer L. Hellier
1 2 3 4 5 Chua HC, Chebib M (2017). GABAA Receptors and the Diversity in their Structure and Pharmacology. Advances in Pharmacology (San Diego, Calif.). Advances in Pharmacology. 79. tr. 1–34. doi:10.1016/bs.apha.2017.03.003. ISBN 9780128104132. PMID 28528665.
↑ Löscher, W.; Rogawski, M. A. (2012). “How theories evolved concerning the mechanism of action of barbiturates”. Epilepsia. 53: 12–25. doi:10.1111/epi.12025. PMID 23205959.
↑ Olsen RW, Betz H (2006). “GABA and glycine”. Trong Siegel GJ, Albers RW, Brady S, Price DD (biên tập). Basic Neurochemistry: Molecular, Cellular and Medical Aspects (ấn bản 7). Elsevier. tr. 291–302. ISBN 978-0-12-088397-4.
↑ (a) Herd MB, Belelli D, Lambert JJ (tháng 10 năm 2007). “Neurosteroid modulation of synaptic and extrasynaptic GABA(A) receptors”. Pharmacology & Therapeutics. 116 (1): 20–34. doi:10.1016/j.pharmthera.2007.03.007. PMID 17531325.; (b) Hosie AM, Wilkins ME, da Silva HM, Smart TG (tháng 11 năm 2006). “Endogenous neurosteroids regulate GABAA receptors through two discrete transmembrane sites”. Nature. 444 (7118): 486–9. doi:10.1038/nature05324. PMID 17108970.; (c)Agís-Balboa RC, Pinna G, Zhubi A, Maloku E, Veldic M, Costa E, Guidotti A (tháng 9 năm 2006). “Characterization of brain neurons that express enzymes mediating neurosteroid biosynthesis”. Proceedings of the National Academy of Sciences of the United States of America. 103 (39): 14602–7. doi:10.1073/pnas.0606544103. PMC 1600006. PMID 16984997.; (d) Akk G, Shu HJ, Wang C, Steinbach JH, Zorumski CF, Covey DF, Mennerick S (tháng 12 năm 2005). “Neurosteroid access to the GABAA receptor”. The Journal of Neuroscience. 25 (50): 11605–13. doi:10.1523/JNEUROSCI.4173-05.2005. PMID 16354918.; (e) Belelli D, Lambert JJ (tháng 7 năm 2005). “Neurosteroids: endogenous regulators of the GABA(A) receptor”. Nature Reviews. Neuroscience. 6 (7): 565–75. doi:10.1038/nrn1703. PMID 15959466.; (f) Pinna G, Costa E, Guidotti A (tháng 6 năm 2006). “Fluoxetine and norfluoxetine stereospecifically and selectively increase brain neurosteroid content at doses that are inactive on 5-HT reuptake”. Psychopharmacology. 186 (3): 362–72. doi:10.1007/s00213-005-0213-2. PMID 16432684.; (g) Dubrovsky BO (tháng 2 năm 2005). “Steroids, neuroactive steroids and neurosteroids in psychopathology”. Progress in Neuro-Psychopharmacology & Biological Psychiatry. 29 (2): 169–92. doi:10.1016/j.pnpbp.2004.11.001. PMID 15694225.; (h) Mellon SH, Griffin LD (2002). “Neurosteroids: biochemistry and clinical significance”. Trends in Endocrinology and Metabolism. 13 (1): 35–43. doi:10.1016/S1043-2760(01)00503-3. PMID 11750861.; (i) Puia G, Santi MR, Vicini S, Pritchett DB, Purdy RH, Paul SM, Seeburg PH, Costa E (tháng 5 năm 1990). “Neurosteroids act on recombinant human GABAA receptors”. Neuron. 4 (5): 759–65. doi:10.1016/0896-6273(90)90202-Q. PMID 2160838.; (j) Majewska MD, Harrison NL, Schwartz RD, Barker JL, Paul SM (tháng 5 năm 1986). “Steroid hormone metabolites are barbiturate-like modulators of the GABA receptor”. Science. 232 (4753): 1004–7. doi:10.1126/science.2422758. PMID 2422758.; (k) Reddy DS, Rogawski MA (2012). “Neurosteroids — Endogenous Regulators of Seizure Susceptibility and Role in the Treatment of Epilepsy”. Trong Noebels JL, Avoli M, Rogawski MA, và đồng nghiệp (biên tập). Jasper's Basic Mechanisms of the Epilepsies [Internet]. 4th edition. Bethesda (MD): National Center for Biotechnology Information (US). National Center for Biotechnology Information (US).
↑ Toraskar, Mrunmayee; Pratima R.P. Singh; Shashank Neve (2010). “STUDY OF GABAERGIC AGONISTS” (PDF). Deccan Journal of Pharmacology. 1 (2): 56–69. Bản gốc (PDF) lưu trữ ngày 16 tháng 10 năm 2013. Truy cập ngày 25 tháng 8 năm 2019.
↑ Vanlersberghe, C; Camu, F (2008). Etomidate and other non-barbiturates. Handbook of Experimental Pharmacology. 182. tr. 267–82. doi:10.1007/978-3-540-74806-9_13. ISBN 978-3-540-72813-9. PMID 18175096.
↑ Dzitoyeva S, Dimitrijevic N, Manev H (2003). “γ-aminobutyric acid B receptor 1 mediates behavior-impairing actions of alcohol in Drosophila: adult RNA interference and pharmacological evidence”. Proc. Natl. Acad. Sci. U.S.A. 100 (9): 5485–5490. Bibcode:2003PNAS..100.5485D. doi:10.1073/pnas.0830111100. PMC 154371. PMID 12692303.
↑ Mihic SJ, Ye Q, Wick MJ, Koltchine VV, Krasowski MD, Finn SE, Mascia MP, Valenzuela CF, Hanson KK, Greenblatt EP, Harris RA, Harrison NL (1997). “Sites of alcohol and volatile anaesthetic action on GABAA and glycine receptors”. Nature. 389 (6649): 385–389. Bibcode:1997Natur.389..385M. doi:10.1038/38738. PMID 9311780.
↑ Boehm SL, Ponomarev I, Blednov YA, Harris RA (2006). “From gene to behavior and back again: new perspectives on GABAAreceptor subunit selectivity of alcohol actions”. Adv. Pharmacol. 54 (8): 1581–1602. doi:10.1016/j.bcp.2004.07.023. PMID 17175815.
↑ Fisher JL (tháng 1 năm 2009). “The anti-convulsant stiripentol acts directly on the GABA(A) receptor as a positive allosteric modulator”. Neuropharmacology. 56 (1): 190–7. doi:10.1016/j.neuropharm.2008.06.004. PMC 2665930. PMID 18585399.
↑ Ueno, S; Bracamontes, J; Zorumski, C; Weiss, DS; Steinbach, JH (1997). “Bicuculline and gabazine are allosteric inhibitors of channel opening of the GABAA receptor”. The Journal of Neuroscience. 17 (2): 625–34. doi:10.1523/jneurosci.17-02-00625.1997. PMID 8987785.
↑ Olsen RW (tháng 4 năm 2000). “Absinthe and gamma-aminobutyric acid receptors”. Proc. Natl. Acad. Sci. U.S.A. 97 (9): 4417–8. doi:10.1073/pnas.97.9.4417. PMC 34311. PMID 10781032.
↑ Whitwam, J. G.; Amrein, R. (ngày 1 tháng 1 năm 1995). “Pharmacology of flumazenil”. Acta Anaesthesiologica Scandinavica. Supplementum. 108: 3–14. ISSN 0515-2720. PMID 8693922.
↑ “Chloroacetaldehydes”, Bách khoa toàn thư Ullmann về Hóa chất công nghiệp, Weinheim: Wiley-VCH, 2005, doi:10.1002/14356007.a06_527.pub2
↑ Lu, J.; Greco, M. A. (2006). “Sleep circuitry and the hypnotic mechanism of GABAA drugs”. Journal of Clinical Sleep Medicine. 2 (2): S19–S26. PMID 17557503.
↑ Singh YN, Singh NN (2002). “Therapeutic potential of kava in the treatment of anxiety disorders”. CNS Drugs. 16 (11): 731–43. doi:10.2165/00023210-200216110-00002. PMID 12383029.
↑ Dimitrijevic N, Dzitoyeva S, Satta R, Imbesi M, Yildiz S, Manev H (2005). “Drosophila GABAB receptors are involved in behavioral effects of gamma-hydroxybutyric acid (GHB)”. Eur. J. Pharmacol. 519 (3): 246–252. doi:10.1016/j.ejphar.2005.07.016. PMID 16129424.
↑ Awad R, Muhammad A, Durst T, Trudeau VL, Arnason JT (tháng 8 năm 2009). “Bioassay-guided fractionation of lemon balm (Melissa officinalis L.) using an in vitro measure of GABA transaminase activity”. Phytother Res. 23 (8): 1075–81. doi:10.1002/ptr.2712. PMID 19165747.
↑ Celikyurt IK, Mutlu O, Ulak G, Akar FY, Erden F (2011). “Gabapentin, A GABA analogue, enhances cognitive performance in mice”. Neuroscience Letters. 492 (2): 124–8. Bibcode:2006NeuL..400..197D. doi:10.1016/j.neulet.2011.01.072. PMID 21296127.
↑ Park DH, Mirabella R, Bronstein PA, Preston GM, Haring MA, Lim CK, Collmer A, Schuurink RC (tháng 10 năm 2010). “Mutations in γ-aminobutyric acid (GABA) transaminase genes in plants or Pseudomonas syringae reduce bacterial virulence”. Plant J. 64 (2): 318–30. doi:10.1111/j.1365-313X.2010.04327.x. PMID 21070411.
↑ Bouché N, Fromm H (tháng 3 năm 2004). “GABA in plants: just a metabolite?”. Trends Plant Sci. 9 (3): 110–5. doi:10.1016/j.tplants.2004.01.006. PMID 15003233.
↑ Roberts MR (tháng 9 năm 2007). “Does GABA Act as a Signal in Plants?: Hints from Molecular Studies”. Plant Signal Behav. 2 (5): 408–9. doi:10.4161/psb.2.5.4335. PMC 2634229. PMID 19704616.